Memory formation under acute stress

Using a mouse model, researchers found that blockade of cannabinoid type-1 receptors on cells that express the enzyme dopamine β-hydroxylase and produce the neurotransmitters adrenaline or noradrenaline in the peripheral and the central nervous systems prevents the impairment of nonemotional memory formation that occurs during acute stress, findings that may have implications for the treatment of cognitive alterations in stress-related disorders. - Read at

Article #15-25066: “Peripheral and central CB1 cannabinoid receptors control stress-induced impairment of memory consolidation,” by Arnau Busquets-Garcia et al.